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Dr. Rashid A. Buttar Now Helping Desiree Jennings
Medical Director, Dr. Rashid A. Buttar is a graduate of the University of Osteopathic Medicine and Health Sciences, College of Medicine and Surgery. He trained in General Surgery and Emergency Medicine and served as Brigade Surgeon and Director of Emergency Medicine while serving in the U.S. Army. Dr. Buttar is board certified in Clinical Metal Toxicology, Preventive Medicine, is board eligible in Emergency Medicine and has achieved fellowship status in three separate medical societies.
The Evil War Against Alternative Medicine And Who's Behind It!
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10/13/2009:
Desiree Jennings - The Flu, & a 'Shot to the System'
Dystonia: "While sounding like a fictional land from a J. R. R. Tolkien novel, Dystonia is a neurological movement disorder where sustained muscle contractions cause body jerks, and abnormal or repetitive movements. The disorder may be inherited or caused by other factors such as birth-related or other physical trauma, infection, poisoning, or reaction to drugs, particularly neuroleptics - according to the National Institute of Neurological Disorders and Stroke."
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What is Dystonia?
Dystonia Protein Linked to Problem Common in Other Neurological Disorders
A new study links the protein that is impaired in the movement disorder torsion dystonia to a problem that is common to many neurological diseases. The finding may point to new treatments for dystonia, Parkinson's disease, and other disorders.
In people with torsion dystonia, an altered gene causes a protein called torsinA to be formed in an abnormal way. Researchers have long wondered how the abnormal version of the protein leads to the disease. The new study shows that the normal protein helps to prevent abnormally folded proteins from clumping together, or aggregating, inside the body's cells. Abnormal clumps of proteins have been identified in Parkinson's, Alzheimer's, Huntington's, and a variety of other neurological diseases. The study appears in the February 1, 2003, issue of Human Molecular Genetics. 1
[ Is it not possible that the "altered gene" which triggers Dystonia in humans
can enter the human body via the influenza or swine flu vaccine? Since different viruses
are designed, altered and/or genetically manipulated in the lab is it not possible that the
"altered gene" which triggers dystonia in people originated IN a pharmaceutical industry lab? Is it not possible that the "altered gene" is *IN* the vaccine which is injected into people? ]
Dystonias are a group of movement disorders in which sustained muscle contractions cause twisting and repetitive movements or abnormal postures. Torsion dystonia is the most severe early-onset form of dystonia. Symptoms usually begin around age 12. While the disorder typically begins in just one part of the body, it eventually spreads to other regions and can leave individuals severely disabled and confined to a wheelchair.
Much of the way proteins work depends on their shape. However, proteins are complex molecules and they often fold into abnormal shapes. Abnormally folded proteins tend to clump together in ways that may be harmful to the cell. One misfolded protein can also cause others to become misfolded.
"It's like clogging a drain," says lead investigator Guy Caldwell, Ph.D., of The University of Alabama at Tuscaloosa. "All it takes is a couple of hairs to attract others, and soon the drain can't function correctly." The protein clumps may prevent cells from carrying out normal activities. They also may make the cells less resistant to stress-related damage, he adds. Scientists have identified several proteins called chaperones that help to prevent other proteins from folding incorrectly and clumping together.
Source of above information about Dystonias:
http://www.ninds.nih.gov/news_and_events/news_articles/news_article_torsion_dystonia.htm
What is Guillain-Barré Syndrome?
No one yet knows why Guillain-Barré—which is not contagious—strikes some people and not others. Nor does anyone know exactly what sets the disease in motion.
[ Squalene, which is found in various flu vaccines, if injected into the human body can trigger Guillain-Barré Syndrome! They know that! They just don't want to state what they already know! ]
What scientists do know is that the body's immune system begins to attack the body itself, causing what is known as an autoimmune disease. Usually the cells of the immune system attack only foreign material and invading organisms. In Guillain-Barré syndrome, however, the immune system starts to destroy the myelin sheath that surrounds the axons of many peripheral nerves, or even the axons themselves (axons are long, thin extensions of the nerve cells; they carry nerve signals). The myelin sheath surrounding the axon speeds up the transmission of nerve signals and allows the transmission of signals over long distances.
In diseases in which the peripheral nerves' myelin sheaths are injured or degraded, the nerves cannot transmit signals efficiently. That is why the muscles begin to lose their ability to respond to the brain's commands, commands that must be carried through the nerve network. The brain also receives fewer sensory signals from the rest of the body, resulting in an inability to feel textures, heat, pain, and other sensations. Alternately, the brain may receive inappropriate signals that result in tingling, "crawling-skin," or painful sensations. Because the signals to and from the arms and legs must travel the longest distances they are most vulnerable to interruption. Therefore, muscle weakness and tingling sensations usually first appear in the hands and feet and progress upwards.
When Guillain-Barré is preceded by a viral or bacterial infection, it is possible that the virus has changed the nature of cells in the nervous system so that the immune system treats them as foreign cells. It is also possible that the virus makes the immune system itself less discriminating about what cells it recognizes as its own, allowing some of the immune cells, such as certain kinds of lymphocytes and macrophages, to attack the myelin. Sensitized T lymphocytes cooperate with B lymphocytes to produce antibodies against components of the myelin sheath and may contribute to destruction of the myelin. Scientists are investigating these and other possibilities to find why the immune system goes awry in Guillain-Barré syndrome and other autoimmune diseases. The cause and course of Guillain-Barré syndrome is an active area of neurological investigation, incorporating the cooperative efforts of neurological scientists, immunologists, and virologists.
Source of above Information about Guillain-Barre Syndrome:
http://www.ninds.nih.gov/disorders/gbs/detail_gbs.htm#138143139
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